Hemolytic anemia is a kind of anemia usually caused by an autoimmune reaction, which refers to the lower amount of red blood cells in your blood. With hemolytic anemia, red blood cells are broken down faster before new ones can be produced, and before the end of their lifespan. Recent developments in the field provide strong evidence that hemolytic anemia is an autoimmune disease. By the end of this article the reader will know all the basic facts about this autoimmune disease, as well as some exciting news about a breakthrough alternative protocol that is reported to have an amazing success rate in 1000s of people worldwide.
Hemolytic anemia - How does it manifest?
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Red cells are destroyed at an accelerated and abnormal level The breakdown of hemoglobin is increased, which in turn may cause increases of bilirubin and urobilinogen levels, or other conditions Bone marrow reacts to compensate by expanding The balance of red cell breakdowns and the compensation of the bone marrow determines how severe the anemia will be.
Hemolytic anemia - How to recognize it?
Usually, all of the symptoms of anemia are present, including shortness of breath, fatigue, pallor, and a chance of heart failure). If a lot of bilirubin reaches the biliary tract, it can cause gallstones. Also, if there is a lot of hemoglobin being released, it may cause pressure on the pulmonary artery, or pulmonary hypertension, which, in turn, can cause occasional fainting and chest pain.
Hemolytic anemia - Treatment Options
Treatment used for curing his disease depends on what caused it:
Hemolytic anemia is a controversial disease because of the lack of knowledge in conventional medicine on the causes of the disease. But, lately, some exciting news has been reported on the discoveries of underlying chemical imbalance and causes. Based on this, a new natural treatment protocol called The Norton Protocol is fashioned with amazing reported success rate that offers hope to all the sufferers.
Hemolytic Anemia - All You Need To Know - Exciting News SEVERS DISEASE
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